Search results for "Mitochondrial Swelling"

showing 2 items of 2 documents

Heterogeneity of osteogenesis imperfecta. Biochemical and morphological findings in a case of type III according to Sillence.

1986

A male infant with pale-blue sclerae, who died at the age of 6 weeks through the aspiration of food, presented multiple fractures and deformation of the long tubular bones. The clinical and radiological findings and the course indicated osteogenesis imperfecta, type III, according to Sillence's classification. The family history was unremarkable. Light and electron microscopic studies of iliac crest bone obtained postmortem, showed an abrupt interruption of endochondral ossification, with an active periosteal ossification. In the region of the fractures, a mixed desmochondral callus was seen. The endoplasmic reticulum of the osteoblasts was markedly dilated, the mitochondria were swollen. T…

MaleProlineEndoplasmic ReticulumHydroxylationIliac crestHydroxylysineBone and BonesOsteogenesis Imperfecta Type IIIchemistry.chemical_compoundMedicineHumansAmino AcidsEndochondral ossificationSkinOsteoblastsbusiness.industryOsteoidCartilageInfantAnatomyOsteogenesis Imperfectamedicine.diseaseChromatography Ion ExchangeHydroxylysinemedicine.anatomical_structureCartilagechemistryOsteogenesis imperfectaPediatrics Perinatology and Child HealthCollagenbusinessMitochondrial SwellingReticulumEuropean journal of pediatrics
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Studies on the subcellular pathophysiology of acute lethal cell injury.

1974

Summary In this paper we have summarized the effects of acute lethal injury on the cell. Such injuries are defined as injuries that result in cell death within a relatively short period of time usually minutes or hours. Following death; the cell undergoes necrosis. Ultrastructural and biochemical methods are needed to study pathophysiology. The cell passes through a series of stages numbered 1 through 7. Stages 1 through 4 are reversible while 5 through 7 are irreversible. Injuries resulting in acute cell death and necrosis include direct damage to the cell membrane, for example by antibody and complement or non-penetrating mercurials or interference with mitochondrial energy supply as in i…

Programmed cell deathPathologymedicine.medical_specialtyNecrosisTime FactorsCell SurvivalCellsCellIschemiaMitochondrionBiologyPermeabilityPathology and Forensic MedicineCell Physiological PhenomenaCell membraneKidney Tubules Proximal03 medical and health sciencesNecrosis0302 clinical medicineIschemiamedicineAnimalsHypoxia030304 developmental biology0303 health sciencesCell MembraneGeneral MedicineHypoxia (medical)medicine.diseasePathophysiology3. Good healthMitochondriaRatsMicroscopy Electronmedicine.anatomical_structuremedicine.symptomMitochondrial Swelling030217 neurology & neurosurgeryBeitrage zur Pathologie
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